The tidal breathing, also known as Chen-Sokes respiration, gradually accelerates and deepens from shallow to slow. After reaching the climax, it gradually becomes shallower and slower. After a few seconds of pause, the above state of breathing occurs again, so that the breathing is tidal. Fluctuations. The tidal breathing is characterized by a gradual decrease in breathing and a gradual increase in both cessation and respiration. More common in patients with central nervous system diseases, cerebral circulation disorders and poisoning, the tidal breathing cycle can be as long as 30 seconds to 2 minutes, and the suspension period can last 5 to 30 seconds. It takes a long time to observe this periodic breathing.
It is generally believed that the respiratory center's reactivity to carbon dioxide is reduced, that is, the threshold of respiratory center excitation is higher than normal. The partial pressure of carbon dioxide in the blood is lower than the threshold that excites the respiratory center, thus apnea. When the partial pressure of carbon dioxide in the blood exceeds the normal level and reaches the threshold, the respiratory center can be excited and the breathing can be restored. After a while of breathing, the partial pressure of carbon dioxide in the blood drops below the threshold level, and the respiratory center stops moving and the breathing stops.
Therefore, clinically, patients with tidal breathing should be examined:
First, physical examination
Taking a medical history gives us a first impression and revelation, and also guides us to a concept of the nature of the disease.
Second, laboratory inspection
Laboratory examinations must be summarized and analyzed based on objective data learned from medical history and physical examination, from which several diagnostic possibilities may be proposed, and further consideration should be given to those examinations to confirm the diagnosis. Such as: peak expiratory flow (PEFR).
Stop breathing is also called Biots breathing. After a regular uniform breathing for a few times, stop for a period of time, and then start breathing evenly, that is, intermittent breathing. This breath is different from tidal breathing. It has the same depth of breathing, instead of undulations. The apnea time is longer than that of tidal breathing, and the number of breaths is also significantly reduced. The mechanism of intermittent breathing is about the same as that of tidal breathing, but the patient breathes. Central inhibition is heavier than tidal breathing, and the condition is more serious. The patient's prognosis is poor, and it often occurs before the breathing completely stops. The disease that causes intermittent breathing is about the same as that of tidal breathing.